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Forsythoside B Protects Cartilage and Subchondral Bone in Osteoarthritis by Regulating the Nrf2/NF-κB Signaling Pathway: Letter to the Editors

  • Meihong Zhang1,
  • Chuanbin Wu1,2,3 and
  • Zhengwei Huang1,3,* 
 Author information 

Dear Editors,

Forsythoside B (FTS-B) is a natural phenylethanoid glycoside isolated from the dried fruit and leaves of Forsythia suspensa (Thunb.) Vahl, a traditional Chinese medicinal herb.1 We recently came across an interesting study published in International Immunopharmacology by Zhang et al.2 The major findings were that FTS-B prevented the degradation of cartilage and subchondral bone in osteoarthritis (OA) through the activation of the nuclear factor erythroid 2-related factor 2 (Nrf2)/heme oxygenase-1 (HO-1) axis and the deactivation of nuclear factor kappa-B (NF-κB) and mitogen-activated protein kinases pathways.2In vitro and in vivo models were established to validate the bioactivity of FTS-B, which revealed positive outcomes. Relevant studies have also confirmed the positive role of the Nrf2–HO-1 signaling pathway in OA.3 We appreciate the authors’ work, which offers new insights into phytochemical-based therapy for OA.

It is believed that the experimental framework of the original research was logically sound, and thus the conclusions were largely convincing. From the standpoint of drug research and development (R&D), we herein propose some further recommendations for the clinical translation of FTS-B-based therapy.

According to the Methods section of the original research, the mouse OA models were constructed on the right knee. Subsequently, different dosages of FTS-B were administered via intraperitoneal injection. We note that the expected therapeutic effects were observed, but the selection of the administration route can be reconsidered. OA can be regarded as a local disease, while intraperitoneal injection is commonly intended for systemic administration.4 During the future R&D process, the FTS-B formulations for intraperitoneal injection in mice may be converted into intravenous injection for human use. It should be noted that systemic exposure of FTS-B, such as through conventional oral and intravenous drug delivery methods, may lead to adverse effects or even toxicities, including potential risks arising from its extensive distribution in non-target organs, as well as possible pseudoallergic reactions.5

To mitigate this issue, one may choose intra-articular injection for FTS-B, which is pertinent to local drug delivery.6 In this way, FTS-B can concentrate at the OA lesion site, and the systemic toxicity can be reduced. Moreover, some low-pain or painless injection techniques are currently available for intra-articular injection, which can improve patient compliance. Most importantly, FTS-B possesses a moderate molecular weight (C34H44O19) and favorable water solubility, suggesting a potential physicochemical basis for its formulation as an intra-articular injection solution and initially supporting its compatibility with low-pain injection technologies. For instance, intelligent insulin pens, as mature and portable injection devices, alleviate instantaneous pain during puncture through their ultra-fine needles.7 Patients can master self-administration at home with simple guidance and perform regular injections independently, significantly lowering the threshold for use. Microneedle technology utilizes arrayed microneedles with diameters ranging from tens to hundreds of micrometers to penetrate the skin and superficial tissues surrounding the joint.8 By avoiding regions dense with nerve endings, microneedles achieve a near-painless effect. However, it is noteworthy that insulin pens and microneedles are mostly designed for shallow joint injections, not for deep joint delivery. We anticipate that low-pain/painless intra-articular injection is particularly suitable for maintenance treatment of patients with mild-to-moderate OA, as well as for patient populations intolerant to systemic administration or sensitive to injection-related pain. However, it should be specifically noted that the therapeutic effects of intra-articular injections are time-limited, with limited short-term efficacy and insufficient long-term symptom relief.9 Additionally, there is a potential risk of inducing systemic allergic adverse reactions.10 Therefore, the compatibility between FTS-B formulations and these techniques must be investigated in depth.

Although this letter aims to provide some suggestions for R&D considerations, the findings of the original research are sufficiently significant. We thank the authors for demonstrating the potential value of FTS-B in OA management.

Declarations

Acknowledgement

None.

Funding

Not applicable.

Conflict of interest

The authors have no conflict of interest related to this publication.

Authors’ contributions

Study concept and design, drafting of the manuscript (MZ), critical revision of the manuscript for important intellectual content, and study supervision (CW, ZH). All authors have made significant contributions to this study and approved the final manuscript.

References

  1. Wang Y, Chen Y, Lu J, Xiao Q, Li G, Wang R, et al. Forsythoside B ameliorates neuroinflammation via inhibiting NLRP3 inflammasome of glial cells in experimental autoimmune encephalomyelitis mice. Brain Res Bull 2025;220:111182 View Article PubMed/NCBI
  2. Zhang H, He Z, Wang H, Lu F, Chen H. Forsythoside B protects cartilage and subchondral bone in osteoarthritis by regulating the Nrf2/NF - κ B signaling pathway. Int Immunopharmacol 2026;168(Pt 1):115762 View Article PubMed/NCBI
  3. Liu YS, Zhong HB, Liu WL, He XH, Zhan XR, Sun CH. Icariin alleviates the apoptosis of chondrocytes in osteoarthritis through regulating SIRT-1-Nrf2-HO-1 signaling. Chem Biol Drug Des 2024;103(4):e14518 View Article PubMed/NCBI
  4. Yang Y, Hao C, Jiao T, Yang Z, Li H, Zhang Y, et al. Osteoarthritis treatment via the GLP-1-mediated gut-joint axis targets intestinal FXR signaling. Science 2025;388(6742):eadt0548 View Article PubMed/NCBI
  5. Han J, Zhang Y, Pan C, Xian Z, Pan C, Zhao Y, et al. Forsythoside A and Forsythoside B Contribute to Shuanghuanglian Injection-Induced Pseudoallergic Reactions through the RhoA/ROCK Signaling Pathway. Int J Mol Sci 2019;20(24):6266 View Article PubMed/NCBI
  6. Mitsou E, Klein J. Liposome-Based Interventions in Knee Osteoarthritis. Small 2025;21(17):e2410060 View Article PubMed/NCBI
  7. Masierek M, Nabrdalik K, Janota O, Kwiendacz H, Macherski M, Gumprecht J. The Review of Insulin Pens-Past, Present, and Look to the Future. Front Endocrinol (Lausanne) 2022;13:827484 View Article PubMed/NCBI
  8. Zhao P, Zhou Z, Wolter T, Womelsdorf J, Somers A, Feng Y, et al. Engineering microneedles for biosensing and drug delivery. Bioact Mater 2025;52:36-59 View Article PubMed/NCBI
  9. Benzon HT, Provenzano DA, Nagpal A, Souza D, Eckmann MS, Nelson AM, et al. Use and safety of corticosteroid injections in joints and musculoskeletal soft tissue: guidelines from the American Society of Regional Anesthesia and Pain Medicine, the American Academy of Pain Medicine, the American Society of Interventional Pain Physicians, and the International Pain and Spine Intervention Society. Reg Anesth Pain Med 2025 View Article PubMed/NCBI
  10. Ijsselmuiden OE, Knegt-Junk KJ, van Wijk RG, van Joost T. Cutaneous adverse reactions after intra-articular injection of triamcinolone acetonide. Acta Derm Venereol 1995;75(1):57-58 View Article PubMed/NCBI

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Zhang M, Wu C, Huang Z. Forsythoside B Protects Cartilage and Subchondral Bone in Osteoarthritis by Regulating the Nrf2/NF-κB Signaling Pathway: Letter to the Editors. Explor Res Hypothesis Med. 2026;11(2):e00063. doi: 10.14218/ERHM.2025.00063.
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Article History
Received Revised Accepted Published
November 11, 2025 February 9, 2026 March 23, 2026 April 9, 2026
DOI http://dx.doi.org/10.14218/ERHM.2025.00063
  • Exploratory Research and Hypothesis in Medicine
  • pISSN 2993-5113
  • eISSN 2472-0712
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Forsythoside B Protects Cartilage and Subchondral Bone in Osteoarthritis by Regulating the Nrf2/NF-κB Signaling Pathway: Letter to the Editors

Meihong Zhang, Chuanbin Wu, Zhengwei Huang
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